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2520

H pylori and Gastric Cancer - Updates in Pathogenesis and Diagnosis

Date
May 18, 2024

SOCIETY: AGA Mechanisms underlying H pylori related gastric pathology

Presentations:

IL-17 SIGNALING REDUCES GASTRIC CARCINOGENESIS BY ALTERING FUNCTION OF THE HELICOBACTER CAG TYPE 4 SECRETION SYSTEM AND SUPPRESSING LRIG1 ACTIVATION
TARGETED IN VIVO COMPETITION AMONG CO-INFECTING HELICOBACTER PYLORI STRAINS INCREASES CAGA-DEPENDENT INFLAMMATION, INJURY, AND CARCINOGENESIS WITHIN THE CONTEXT OF IRON DEFICIENCY
IMPLICATION OF HYPUSINATION IN HELICOBACTER PYLORI-INDUCED GASTRIC CARCINOGENESIS
HELICOBACTER PYLORI ERADICATION AND GASTRIC CANCER RISK IN GASTRIC ULCER PATIENTS: A NATIONWIDE COHORT STUDY
HELICOBACTER PYLORI-INDUCED FERROPTOSIS CONTRIBUTES TO ATROPHIC GASTRITIS VIA MODULATING SELENIUM UTILIZATION BY GPX4.

Moderators

Speaker Image for Steven Moss
Brown University
Speaker Image for John Kao
University of Michigan

Related Products

Thumbnail for IL-17 SIGNALING REDUCES GASTRIC CARCINOGENESIS  BY ALTERING FUNCTION OF THE HELICOBACTER CAG TYPE 4 SECRETION SYSTEM AND SUPPRESSING LRIG1 ACTIVATION
IL-17 SIGNALING REDUCES GASTRIC CARCINOGENESIS BY ALTERING FUNCTION OF THE HELICOBACTER CAG TYPE 4 SECRETION SYSTEM AND SUPPRESSING LRIG1 ACTIVATION
_Helicobacter pylori_ is the strongest risk factor for gastric cancer; however, only a minority of infected individuals develop disease. One microbial cancer-linked locus is the _cag_ type 4 secretion system (_cag_T4SS), which translocates the oncoprotein CagA into host cells…
Thumbnail for PANEL DISCUSSION
PANEL DISCUSSION
40 years after the organism was first recognized and grown in culture, Helicobacter pylori infections are still commonly encountered in practice,. This symposium will focus on several areas that remain unresolved to this day - including how best to manage H…